A role for ataxia telangiectasia mutated in insulin-independent stimulation of glucose transport

Literature reports suggest that ataxia telangiectasia mutated (ATM) can activate the AMP-activated protein kinase (AMPK), a protein that can stimulate glucose transport in skeletal muscle. We hypothesized that 5-aminoimidazole-4-carboxamide ribonucleotide (AICAR), an AMPK activator, would increase g...

पूर्ण विवरण

में बचाया:
ग्रंथसूची विवरण
में प्रकाशित:Trends Cell Mol Biol
मुख्य लेखकों: Spears, Larry D., Renth, Allyson L., McKuin, Michael R., Kennedy, Anne R., Andrisse, Stanley, Briggs, Nell E., Fisher, Jonathan S.
स्वरूप: Artigo
भाषा:Inglês
प्रकाशित: 2017
विषय:
Article
ऑनलाइन पहुंच:https://ncbi.nlm.nih.gov/pmc/articles/PMC6287632/
https://ncbi.nlm.nih.gov/pubmed/30542240
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