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A role for ataxia telangiectasia mutated in insulin-independent stimulation of glucose transport

Literature reports suggest that ataxia telangiectasia mutated (ATM) can activate the AMP-activated protein kinase (AMPK), a protein that can stimulate glucose transport in skeletal muscle. We hypothesized that 5-aminoimidazole-4-carboxamide ribonucleotide (AICAR), an AMPK activator, would increase g...

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Publié dans:Trends Cell Mol Biol
Auteurs principaux: Spears, Larry D., Renth, Allyson L., McKuin, Michael R., Kennedy, Anne R., Andrisse, Stanley, Briggs, Nell E., Fisher, Jonathan S.
Format: Artigo
Langue:Inglês
Publié: 2017
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Accès en ligne:https://ncbi.nlm.nih.gov/pmc/articles/PMC6287632/
https://ncbi.nlm.nih.gov/pubmed/30542240
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