Critical role of mitosis in spontaneous late-onset Alzheimer’s disease; from a Shugoshin 1 cohesinopathy mouse model

From early-onset Alzheimer’s disease (EOAD) studies, the amyloid-beta hypothesis emerged as the foremost theory of the pathological causes of AD. However, how amyloid-beta accumulation is triggered and progresses toward senile plaques in spontaneous late-onset Alzheimer’s disease (LOAD) in humans re...

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Bibliographische Detailangaben
Veröffentlicht in:Cell Cycle
Hauptverfasser: Rao, Chinthalapally V., Farooqui, Mudassir, Asch, Adam S., Yamada, Hiroshi Y.
Format: Artigo
Sprache:Inglês
Veröffentlicht: Taylor & Francis 2018
Schlagworte:
Review
Online Zugang:https://ncbi.nlm.nih.gov/pmc/articles/PMC6237434/
https://ncbi.nlm.nih.gov/pubmed/30231670
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1080/15384101.2018.1515554
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