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Critical role of mitosis in spontaneous late-onset Alzheimer’s disease; from a Shugoshin 1 cohesinopathy mouse model
From early-onset Alzheimer’s disease (EOAD) studies, the amyloid-beta hypothesis emerged as the foremost theory of the pathological causes of AD. However, how amyloid-beta accumulation is triggered and progresses toward senile plaques in spontaneous late-onset Alzheimer’s disease (LOAD) in humans re...
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| Publicado no: | Cell Cycle |
|---|---|
| Main Authors: | , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
Taylor & Francis
2018
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6237434/ https://ncbi.nlm.nih.gov/pubmed/30231670 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1080/15384101.2018.1515554 |
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