Variation in TMEM106B in chronic traumatic encephalopathy

The genetic basis of chronic traumatic encephalopathy (CTE) is poorly understood. Variation in transmembrane protein 106B (TMEM106B) has been associated with enhanced neuroinflammation during aging and with TDP-43-related neurodegenerative disease, and rs3173615, a missense coding SNP in TMEM106B, h...

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Bibliografiska uppgifter
I publikationen:Acta Neuropathol Commun
Huvudupphovsmän: Cherry, Jonathan D., Mez, Jesse, Crary, John F., Tripodis, Yorghos, Alvarez, Victor E., Mahar, Ian, Huber, Bertrand R., Alosco, Michael L., Nicks, Raymond, Abdolmohammadi, Bobak, Kiernan, Patrick T., Evers, Laney, Svirsky, Sarah, Babcock, Katharine, Gardner, Hannah M., Meng, Gaoyuan, Nowinski, Christopher J., Martin, Brett M., Dwyer, Brigid, Kowall, Neil W., Cantu, Robert C., Goldstein, Lee E., Katz, Douglas I., Stern, Robert A., Farrer, Lindsay A., McKee, Ann C., Stein, Thor D.
Materialtyp: Artigo
Språk:Inglês
Publicerad: BioMed Central 2018
Ämnen:
Research
Länkar:https://ncbi.nlm.nih.gov/pmc/articles/PMC6215686/
https://ncbi.nlm.nih.gov/pubmed/30390709
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/s40478-018-0619-9
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