Variation in TMEM106B in chronic traumatic encephalopathy

The genetic basis of chronic traumatic encephalopathy (CTE) is poorly understood. Variation in transmembrane protein 106B (TMEM106B) has been associated with enhanced neuroinflammation during aging and with TDP-43-related neurodegenerative disease, and rs3173615, a missense coding SNP in TMEM106B, h...

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Bibliografiset tiedot
Julkaisussa:Acta Neuropathol Commun
Päätekijät: Cherry, Jonathan D., Mez, Jesse, Crary, John F., Tripodis, Yorghos, Alvarez, Victor E., Mahar, Ian, Huber, Bertrand R., Alosco, Michael L., Nicks, Raymond, Abdolmohammadi, Bobak, Kiernan, Patrick T., Evers, Laney, Svirsky, Sarah, Babcock, Katharine, Gardner, Hannah M., Meng, Gaoyuan, Nowinski, Christopher J., Martin, Brett M., Dwyer, Brigid, Kowall, Neil W., Cantu, Robert C., Goldstein, Lee E., Katz, Douglas I., Stern, Robert A., Farrer, Lindsay A., McKee, Ann C., Stein, Thor D.
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: BioMed Central 2018
Aiheet:
Research
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC6215686/
https://ncbi.nlm.nih.gov/pubmed/30390709
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/s40478-018-0619-9
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