Variation in TMEM106B in chronic traumatic encephalopathy

The genetic basis of chronic traumatic encephalopathy (CTE) is poorly understood. Variation in transmembrane protein 106B (TMEM106B) has been associated with enhanced neuroinflammation during aging and with TDP-43-related neurodegenerative disease, and rs3173615, a missense coding SNP in TMEM106B, h...

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Foilsithe in:Acta Neuropathol Commun
Main Authors: Cherry, Jonathan D., Mez, Jesse, Crary, John F., Tripodis, Yorghos, Alvarez, Victor E., Mahar, Ian, Huber, Bertrand R., Alosco, Michael L., Nicks, Raymond, Abdolmohammadi, Bobak, Kiernan, Patrick T., Evers, Laney, Svirsky, Sarah, Babcock, Katharine, Gardner, Hannah M., Meng, Gaoyuan, Nowinski, Christopher J., Martin, Brett M., Dwyer, Brigid, Kowall, Neil W., Cantu, Robert C., Goldstein, Lee E., Katz, Douglas I., Stern, Robert A., Farrer, Lindsay A., McKee, Ann C., Stein, Thor D.
Formáid: Artigo
Teanga:Inglês
Foilsithe: BioMed Central 2018
Ábhair:
Research
Rochtain Ar Líne:https://ncbi.nlm.nih.gov/pmc/articles/PMC6215686/
https://ncbi.nlm.nih.gov/pubmed/30390709
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/s40478-018-0619-9
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