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Mitochondrial dysfunction reduces the activity of KIR2.1 K(+) channel in myoblasts via impaired oxidative phosphorylation
Myoblast fusion depends on mitochondrial integrity and intracellular Ca(2+) signaling regulated by various ion channels. In this study, we investigated the ionic currents associated with [Ca(2+)](i) regulation in normal and mitochondrial DNA-depleted (ρ0) L6 myoblasts. The ρ0 myoblasts showed impair...
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| 出版年: | Korean J Physiol Pharmacol |
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| 主要な著者: | , , , , , , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
The Korean Physiological Society and The Korean Society of Pharmacology
2018
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6205933/ https://ncbi.nlm.nih.gov/pubmed/30402030 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.4196/kjpp.2018.22.6.697 |
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