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Interaction between smoking and ATG16L1(T300A) triggers Paneth cell defects in Crohn’s disease
It is suggested that subtyping of complex inflammatory diseases can be based on genetic susceptibility and relevant environmental exposure (G+E). We propose that using matched cellular phenotypes in human subjects and corresponding preclinical models with the same G+E combinations is useful to this...
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| Publicado en: | J Clin Invest |
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| Autores principales: | , , , , , , , , , , , , , |
| Formato: | Artigo |
| Lenguaje: | Inglês |
| Publicado: |
American Society for Clinical Investigation
2018
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| Materias: | |
| Acceso en línea: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6205411/ https://ncbi.nlm.nih.gov/pubmed/30137026 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1172/JCI120453 |
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