Synthetic Fragments of Receptor for Advanced Glycation End Products Bind Beta-Amyloid 1–40 and Protect Primary Brain Cells From Beta-Amyloid Toxicity

Receptor for advanced glycation end products (RAGE) is involved in the pathogenesis of Alzheimer’s disease. We have previously revealed that RAGE fragment sequence (60–76) and its shortened analogs sequence (60–70) and (60–65) under intranasal insertion were able to restore memory and improve morpho...

תיאור מלא

שמור ב:
מידע ביבליוגרפי
הוצא לאור ב:Front Neurosci
Main Authors: Kamynina, Anna V., Esteras, Noemi, Koroev, Dmitriy O., Bobkova, Natalia V., Balasanyants, Samson M., Simonyan, Ruben A., Avetisyan, Armine V., Abramov, Andrey Y., Volpina, Olga M.
פורמט: Artigo
שפה:Inglês
יצא לאור: Frontiers Media S.A. 2018
נושאים:
Neuroscience
גישה מקוונת:https://ncbi.nlm.nih.gov/pmc/articles/PMC6170785/
https://ncbi.nlm.nih.gov/pubmed/30319347
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3389/fnins.2018.00681
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