A combination antioxidant therapy to inhibit NOX2 and activate Nrf2 decreases secondary brain damage and improves functional recovery after traumatic brain injury

Uncontrolled oxidative stress contributes to the secondary neuronal death that promotes long-term neurological dysfunction following traumatic brain injury (TBI). Surprisingly, both NADPH oxidase 2 (NOX2) that increases and transcription factor Nrf2 that decreases reactive oxygen species (ROS) are i...

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Bibliografiske detaljer
Udgivet i:J Cereb Blood Flow Metab
Main Authors: Chandran, Raghavendar, Kim, TaeHee, Mehta, Suresh L, Udho, Eshwar, Chanana, Vishal, Cengiz, Pelin, Kim, HwuiWon, Kim, Chanul, Vemuganti, Raghu
Format: Artigo
Sprog:Inglês
Udgivet: SAGE Publications 2017
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Original Articles
Online adgang:https://ncbi.nlm.nih.gov/pmc/articles/PMC6168911/
https://ncbi.nlm.nih.gov/pubmed/29083257
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1177/0271678X17738701
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