A combination antioxidant therapy to inhibit NOX2 and activate Nrf2 decreases secondary brain damage and improves functional recovery after traumatic brain injury

Uncontrolled oxidative stress contributes to the secondary neuronal death that promotes long-term neurological dysfunction following traumatic brain injury (TBI). Surprisingly, both NADPH oxidase 2 (NOX2) that increases and transcription factor Nrf2 that decreases reactive oxygen species (ROS) are i...

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Bibliografiska uppgifter
I publikationen:J Cereb Blood Flow Metab
Huvudupphovsmän: Chandran, Raghavendar, Kim, TaeHee, Mehta, Suresh L, Udho, Eshwar, Chanana, Vishal, Cengiz, Pelin, Kim, HwuiWon, Kim, Chanul, Vemuganti, Raghu
Materialtyp: Artigo
Språk:Inglês
Publicerad: SAGE Publications 2017
Ämnen:
Original Articles
Länkar:https://ncbi.nlm.nih.gov/pmc/articles/PMC6168911/
https://ncbi.nlm.nih.gov/pubmed/29083257
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1177/0271678X17738701
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