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Oxidation of KCNB1 channels in the human brain and in mouse model of Alzheimer’s disease

Oxidative modification of the voltage-gated K(+) channel subfamily B member 1 (KCNB1, Kv2.1) is emerging as a mechanism of neuronal vulnerability potentially capable of affecting multiple conditions associated with oxidative stress, from normal aging to neurodegenerative disease. In this study we re...

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Detalhes bibliográficos
Publicado no:Cell Death Dis
Main Authors: Wei, Yu, Shin, Mi Ryung, Sesti, Federico
Formato: Artigo
Idioma:Inglês
Publicado em: Nature Publishing Group UK 2018
Assuntos:
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC6062629/
https://ncbi.nlm.nih.gov/pubmed/30050035
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/s41419-018-0886-1
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