Persistent 3′-phosphate termini and increased cytotoxicity of radiomimetic DNA double-strand breaks in cells lacking polynucleotide kinase/phosphatase despite presence of an alternative 3′-phosphatase

Lignende værker

• TDP1 suppresses mis-joining of radiomimetic DNA double-strand breaks and cooperates with Artemis to promote optimal nonhomologous end joining
  af: Kawale, Ajinkya S, et al.
  Udgivet: (2018)
• Patching and Single-Strand Ligation in Nonhomologous DNA End Joining Despite Persistence of a Closely Opposed 3′-Phosphoglycolate-Terminated Strand Break
  af: Zhou, Rui-Zhe, et al.
  Udgivet: (2010)
• Independent mechanisms of stimulation of polynucleotide kinase/phosphatase by phosphorylated and non-phosphorylated XRCC1
  af: Lu, Meiling, et al.
  Udgivet: (2010)
• Mechanism of Action of an Imidopiperidine Inhibitor of Human Polynucleotide Kinase/Phosphatase
  af: Freschauf, Gary K., et al.
  Udgivet: (2010)
• Tracking the processing of damaged DNA double-strand break ends by ligation-mediated PCR: increased persistence of 3′-phosphoglycolate termini in SCAN1 cells
  af: Akopiants, Konstantin, et al.
  Udgivet: (2014)