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β-Adrenergic receptor trafficking, degradation, and cell-surface expression are altered in dermal fibroblasts from hypertrophic scars
Burn trauma elevates catecholamines for up to 2 years and causes hypertrophic scarring. Propranolol, a non-specific ß1, ß2 adrenergic receptor (AR) inverse agonist, counters the hypermetabolic response to elevated catecholamines and may decrease hypertrophic scarring by an unknown mechanism. We inve...
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| Pubblicato in: | J Invest Dermatol |
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| Autori principali: | , , , , |
| Natura: | Artigo |
| Lingua: | Inglês |
| Pubblicazione: |
2018
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| Soggetti: | |
| Accesso online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6021177/ https://ncbi.nlm.nih.gov/pubmed/29476776 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.jid.2018.01.037 |
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