Histone deacetylase inhibition activates transcription factor Nrf2 and protects against cerebral ischemic damage

Interest in histone deacetylase (HDAC)-based therapeutics as a potential treatment for stroke has grown dramatically. The neuroprotection of HDAC inhibition may involve multiple mechanisms, including modulation of transcription factor acetylation independent of histones. The transcription factor Nrf...

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Библиографические подробности
Опубликовано в: :Free Radic Biol Med
Главные авторы: Wang, Bing, Zhu, Xiaolei, Kim, YunTai, Li, Jie, Huang, Siyuan, Saleem, Sofiyan, Li, Rung-chi, Xu, Yun, Dore, Sylvain, Cao, Wangsen
Формат: Artigo
Язык:Inglês
Опубликовано: 2011
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Article
Online-ссылка:https://ncbi.nlm.nih.gov/pmc/articles/PMC6010182/
https://ncbi.nlm.nih.gov/pubmed/22226832
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.freeradbiomed.2011.12.006
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