Histone deacetylase inhibition activates transcription factor Nrf2 and protects against cerebral ischemic damage

Interest in histone deacetylase (HDAC)-based therapeutics as a potential treatment for stroke has grown dramatically. The neuroprotection of HDAC inhibition may involve multiple mechanisms, including modulation of transcription factor acetylation independent of histones. The transcription factor Nrf...

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Detaylı Bibliyografya
Yayımlandı:Free Radic Biol Med
Asıl Yazarlar: Wang, Bing, Zhu, Xiaolei, Kim, YunTai, Li, Jie, Huang, Siyuan, Saleem, Sofiyan, Li, Rung-chi, Xu, Yun, Dore, Sylvain, Cao, Wangsen
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: 2011
Konular:
Article
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC6010182/
https://ncbi.nlm.nih.gov/pubmed/22226832
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.freeradbiomed.2011.12.006
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