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Loss of Zbtb32 in NOD mice does not significantly alter T cell responses.
Background : We previously identified the transcriptional regulator Zbtb32 as a factor that can promote T cell tolerance in the Non-Obese Diabetic (NOD) mouse, a model of Type 1 diabetes. Antigen targeted to DCIR2 (+) dendritic cells (DCs) in vivo inhibited both diabetes and effector T cell expansio...
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| Veröffentlicht in: | F1000Res |
|---|---|
| Hauptverfasser: | , , , , , , |
| Format: | Artigo |
| Sprache: | Inglês |
| Veröffentlicht: |
F1000 Research Limited
2018
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| Schlagworte: | |
| Online Zugang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5909056/ https://ncbi.nlm.nih.gov/pubmed/29707204 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.12688/f1000research.13864.2 |
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