New insights into the role and mechanism of c-Jun-N-terminal kinase signaling in the pathobiology of liver diseases

Ítems similars

• Antcin H Protects Against Acute Liver Injury Through Disruption of the Interaction of c-Jun-N-Terminal Kinase with Mitochondria
  per: Huo, Yazhen, et al.
  Publicat: (2017)
• c-Jun N-terminal Kinase (JNK)-dependent Acute Liver Injury from Acetaminophen or Tumor Necrosis Factor (TNF) Requires Mitochondrial Sab Protein Expression in Mice
  per: Win, Sanda, et al.
  Publicat: (2011)
• JNK mediates mouse liver injury through a novel Sab (SH3BP5) dependent pathway leading to inactivation of intramitochondrial Src
  per: Win, Sanda, et al.
  Publicat: (2016)
• The Regulation of JNK Signaling Pathways in Cell Death through the Interplay with Mitochondrial SAB and Upstream Post-Translational Effects
  per: Win, Sanda, et al.
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• Role of cAMP-responsive Element-binding Protein (CREB)-regulated Transcription Coactivator 3 (CRTC3) in the Initiation of Mitochondrial Biogenesis and Stress Response in Liver Cells
  per: Than, Tin Aung, et al.
  Publicat: (2011)