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Peripheral complement interactions with amyloid β peptide (Aβ) in Alzheimer’s disease: 1. Erythrocyte clearance of Aβ

INTRODUCTION: Although Aβ is cleared from brain to CSF and the peripheral circulation, mechanisms for its removal from blood remain unresolved. Primates have uniquely evolved a highly effective peripheral clearance mechanism for pathogens, immune adherence, in which erythrocyte complement receptor 1...

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מידע ביבליוגרפי
הוצא לאור ב:	Alzheimers Dement
Main Authors:	Brubaker, William D., Crane, Andrés, Johansson, Jenny U., Yen, Kevin, Garfinkel, Kristina, Mastroeni, Diego, Asok, Priya, Bradt, Bonnie, Sabbagh, Marwan, Wallace, Tanya L., Glavis-Bloom, Courtney, Tenner, Andrea J., Rogers, Joseph
פורמט:	Artigo
שפה:	Inglês
יצא לאור:	2017
נושאים:	Article
גישה מקוונת:	https://ncbi.nlm.nih.gov/pmc/articles/PMC5880643/ https://ncbi.nlm.nih.gov/pubmed/28475854 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.jalz.2017.03.010
תגים:	הוספת תג אין תגיות, היה/י הראשונ/ה לתייג את הרשומה!

Peripheral complement interactions with amyloid β peptide (Aβ) in Alzheimer’s disease: 1. Erythrocyte clearance of Aβ

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