Φορτώνει......
Zn(2+)-induced disruption of neuronal mitochondrial function: synergism with Ca(2+), critical dependence upon cytosolic Zn(2+) buffering, and contributions to neuronal injury
Excitotoxic Zn(2+) and Ca(2+) accumulation contributes to neuronal injury after ischemia or prolonged seizures. Synaptically released Zn(2+) can enter postsynaptic neurons via routes including voltage sensitive Ca(2+) channels (VSCC), and, more rapidly, through Ca(2+) permeable AMPA channels. There...
Αποθηκεύτηκε σε:
| Τόπος έκδοσης: | Exp Neurol |
|---|---|
| Κύριοι συγγραφείς: | , |
| Μορφή: | Artigo |
| Γλώσσα: | Inglês |
| Έκδοση: |
2018
|
| Θέματα: | |
| Διαθέσιμο Online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5849513/ https://ncbi.nlm.nih.gov/pubmed/29355498 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.expneurol.2018.01.012 |
| Ετικέτες: |
Προσθήκη ετικέτας
Δεν υπάρχουν, Καταχωρήστε ετικέτα πρώτοι!
|