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Zn(2+)-induced disruption of neuronal mitochondrial function: synergism with Ca(2+), critical dependence upon cytosolic Zn(2+) buffering, and contributions to neuronal injury

Excitotoxic Zn(2+) and Ca(2+) accumulation contributes to neuronal injury after ischemia or prolonged seizures. Synaptically released Zn(2+) can enter postsynaptic neurons via routes including voltage sensitive Ca(2+) channels (VSCC), and, more rapidly, through Ca(2+) permeable AMPA channels. There...

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Detaylı Bibliyografya
Yayımlandı:Exp Neurol
Asıl Yazarlar: Ji, Sung G., Weiss, John H.
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: 2018
Konular:
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC5849513/
https://ncbi.nlm.nih.gov/pubmed/29355498
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.expneurol.2018.01.012
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