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Caspase-1 self-cleavage is an intrinsic mechanism to terminate inflammasome activity
Host-protective caspase-1 activity must be tightly regulated to prevent pathology, but mechanisms controlling the duration of cellular caspase-1 activity are unknown. Caspase-1 is activated on inflammasomes, signaling platforms that facilitate caspase-1 dimerization and autoprocessing. Previous stud...
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| 出版年: | J Exp Med |
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| 主要な著者: | , , , , , , , , , , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
Rockefeller University Press
2018
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5839769/ https://ncbi.nlm.nih.gov/pubmed/29432122 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1084/jem.20172222 |
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