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Sterol oxidation mediates stress-responsive Vms1 translocation to mitochondria
Vms1 translocates to damaged mitochondria in response to stress, whereupon its binding partner, Cdc48, contributes to mitochondrial protein homeostasis. Mitochondrial targeting of Vms1 is mediated by its conserved Mitochondrial Targeting Domain (MTD), which in unstressed conditions is inhibited by i...
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| Vydáno v: | Mol Cell |
|---|---|
| Hlavní autoři: | , , , , , , , |
| Médium: | Artigo |
| Jazyk: | Inglês |
| Vydáno: |
2017
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| Témata: | |
| On-line přístup: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5837041/ https://ncbi.nlm.nih.gov/pubmed/29149595 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.molcel.2017.10.022 |
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