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Advanced glycation end (AGE) product modification of laminin downregulates Kir4.1 in retinal Müller cells
Diabetic retinopathy (DR) is a major cause of adult blindness. Retinal Müller cells maintain water homeostasis and potassium concentration via inwardly rectifying Kir4.1 channels. Accumulation of advanced glycation end products (AGEs) is a major pathologic event in DR. While diabetes leads to a decr...
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| Udgivet i: | PLoS One |
|---|---|
| Main Authors: | , , , , , |
| Format: | Artigo |
| Sprog: | Inglês |
| Udgivet: |
Public Library of Science
2018
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| Fag: | |
| Online adgang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5825079/ https://ncbi.nlm.nih.gov/pubmed/29474462 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1371/journal.pone.0193280 |
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