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Cx26 drives self-renewal in triple-negative breast cancer via interaction with NANOG and focal adhesion kinase
Tumors adapt their phenotypes during growth and in response to therapies through dynamic changes in cellular processes. Connexin proteins enable such dynamic changes during development, and their dysregulation leads to disease states. The gap junction communication channels formed by connexins have...
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| Publicado no: | Nat Commun |
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| Main Authors: | , , , , , , , , , , , , , , , , , , , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
Nature Publishing Group UK
2018
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5805730/ https://ncbi.nlm.nih.gov/pubmed/29422613 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/s41467-018-02938-1 |
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