A carregar...

Cx26 drives self-renewal in triple-negative breast cancer via interaction with NANOG and focal adhesion kinase

Tumors adapt their phenotypes during growth and in response to therapies through dynamic changes in cellular processes. Connexin proteins enable such dynamic changes during development, and their dysregulation leads to disease states. The gap junction communication channels formed by connexins have...

ver descrição completa

Na minha lista:
Detalhes bibliográficos
Publicado no:Nat Commun
Main Authors: Thiagarajan, Praveena S., Sinyuk, Maksim, Turaga, Soumya M., Mulkearns-Hubert, Erin E., Hale, James S., Rao, Vinay, Demelash, Abeba, Saygin, Caner, China, Arnab, Alban, Tyler J., Hitomi, Masahiro, Torre-Healy, Luke A., Alvarado, Alvaro G., Jarrar, Awad, Wiechert, Andrew, Adorno-Cruz, Valery, Fox, Paul L., Calhoun, Benjamin C., Guan, Jun-Lin, Liu, Huiping, Reizes, Ofer, Lathia, Justin D.
Formato: Artigo
Idioma:Inglês
Publicado em: Nature Publishing Group UK 2018
Assuntos:
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC5805730/
https://ncbi.nlm.nih.gov/pubmed/29422613
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/s41467-018-02938-1
Tags: Adicionar Tag
Sem tags, seja o primeiro a adicionar uma tag!