Cx26 drives self-renewal in triple-negative breast cancer via interaction with NANOG and focal adhesion kinase

Tumors adapt their phenotypes during growth and in response to therapies through dynamic changes in cellular processes. Connexin proteins enable such dynamic changes during development, and their dysregulation leads to disease states. The gap junction communication channels formed by connexins have...

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Veröffentlicht in:Nat Commun
Hauptverfasser: Thiagarajan, Praveena S., Sinyuk, Maksim, Turaga, Soumya M., Mulkearns-Hubert, Erin E., Hale, James S., Rao, Vinay, Demelash, Abeba, Saygin, Caner, China, Arnab, Alban, Tyler J., Hitomi, Masahiro, Torre-Healy, Luke A., Alvarado, Alvaro G., Jarrar, Awad, Wiechert, Andrew, Adorno-Cruz, Valery, Fox, Paul L., Calhoun, Benjamin C., Guan, Jun-Lin, Liu, Huiping, Reizes, Ofer, Lathia, Justin D.
Format: Artigo
Sprache:Inglês
Veröffentlicht: Nature Publishing Group UK 2018
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Online Zugang:https://ncbi.nlm.nih.gov/pmc/articles/PMC5805730/
https://ncbi.nlm.nih.gov/pubmed/29422613
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/s41467-018-02938-1
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