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Knockout of mitochondrial voltage-dependent anion channel type 3 increases reactive oxygen species (ROS) levels and alters renal sodium transport
It has been suggested that voltage-dependent anion channels (VDACs) control the release of superoxide from mitochondria. We have previously shown that reactive oxygen species (ROS) such as superoxide (O(2)(˙̄)) and hydrogen peroxide (H(2)O(2)) stimulate epithelial sodium channels (ENaCs) in sodium-t...
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| Опубликовано в: : | J Biol Chem |
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| Главные авторы: | , , , , , , , , , , , |
| Формат: | Artigo |
| Язык: | Inglês |
| Опубликовано: |
American Society for Biochemistry and Molecular Biology
2018
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| Предметы: | |
| Online-ссылка: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5798297/ https://ncbi.nlm.nih.gov/pubmed/29180450 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1074/jbc.M117.798645 |
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