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Knockout of mitochondrial voltage-dependent anion channel type 3 increases reactive oxygen species (ROS) levels and alters renal sodium transport

It has been suggested that voltage-dependent anion channels (VDACs) control the release of superoxide from mitochondria. We have previously shown that reactive oxygen species (ROS) such as superoxide (O(2)(˙̄)) and hydrogen peroxide (H(2)O(2)) stimulate epithelial sodium channels (ENaCs) in sodium-t...

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Библиографические подробности
Опубликовано в: :J Biol Chem
Главные авторы: Zou, Li, Linck, Valerie, Zhai, Yu-Jia, Galarza-Paez, Laura, Li, Linda, Yue, Qiang, Al-Khalili, Otor, Bao, Hui-Fang, Ma, He-Ping, Thai, Tiffany L., Jiao, Jundong, Eaton, Douglas C.
Формат: Artigo
Язык:Inglês
Опубликовано: American Society for Biochemistry and Molecular Biology 2018
Предметы:
Online-ссылка:https://ncbi.nlm.nih.gov/pmc/articles/PMC5798297/
https://ncbi.nlm.nih.gov/pubmed/29180450
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1074/jbc.M117.798645
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