RhoA S-nitrosylation as a regulatory mechanism influencing endothelial barrier function in response to G(+)-bacterial toxins

Disruption of the endothelial barrier in response to Gram positive (G(+)) bacterial toxins is a major complication of acute lung injury (ALI) and can be further aggravated by antibiotics which stimulate toxin release. The integrity of the pulmonary endothelial barrier is mediated by the balance of d...

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Publié dans:Biochem Pharmacol
Auteurs principaux: Chen, F., Wang, Y., Rafikov, R., Haigh, S., Zhi, W.B., Kumar, S., Doulias, P.T., Rafikova, O., Pillich, H., Chakraborty, T., Lucas, R., Verin, A.D., Catravas, J.D., She, J.X., Black, S.M., Fulton, D.J.R.
Format: Artigo
Langue:Inglês
Publié: 2016
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Article
Accès en ligne:https://ncbi.nlm.nih.gov/pmc/articles/PMC5768143/
https://ncbi.nlm.nih.gov/pubmed/28017778
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.bcp.2016.12.014
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