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Mice deficient in the Shmt2 gene have mitochondrial respiration defects and are embryonic lethal

Accumulation of somatic mutations in mitochondrial DNA (mtDNA) has been proposed to be responsible for human aging and age-associated mitochondrial respiration defects. However, our previous findings suggested an alternative hypothesis of human aging—that epigenetic changes but not mutations regulat...

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Podrobná bibliografie
Vydáno v:Sci Rep
Hlavní autoři: Tani, Haruna, Ohnishi, Sakiko, Shitara, Hiroshi, Mito, Takayuki, Yamaguchi, Midori, Yonekawa, Hiromichi, Hashizume, Osamu, Ishikawa, Kaori, Nakada, Kazuto, Hayashi, Jun-Ichi
Médium: Artigo
Jazyk:Inglês
Vydáno: Nature Publishing Group UK 2018
Témata:
On-line přístup:https://ncbi.nlm.nih.gov/pmc/articles/PMC5765156/
https://ncbi.nlm.nih.gov/pubmed/29323231
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/s41598-017-18828-3
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