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Could blocking the formation of amyloid channels rescue Alzheimer's phenotype?
In a most simplified way, we can say that much of the symptomatology that characterizes Alzheimer's disease (AD) can be attributed to a cascade of toxic events initiated by the presence in the interstitial space of the brain of oligomers of the β‐amyloid peptide (Aβ) peptide, a cleavage by‐prod...
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| Veröffentlicht in: | EMBO Mol Med |
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| Hauptverfasser: | , |
| Format: | Artigo |
| Sprache: | Inglês |
| Veröffentlicht: |
John Wiley and Sons Inc.
2017
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| Schlagworte: | |
| Online Zugang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5760883/ https://ncbi.nlm.nih.gov/pubmed/29208637 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.15252/emmm.201708491 |
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