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A proline deletion in IFNAR1 impairs IFN-signaling and underlies increased resistance to tuberculosis in humans
Type I interferons (IFN), best known for their anti-viral functions, have been shown to impair host resistance to intracellular bacteria in mice. However, the precise role of type I IFN signaling in bacterial infection in humans is unclear. Here, we show that genetic variation in the human IFNAR1 ge...
שמור ב:
| הוצא לאור ב: | Nat Commun |
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| Main Authors: | , , , , , , , , , , , , , , , |
| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
Nature Publishing Group UK
2018
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| נושאים: | |
| גישה מקוונת: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5758831/ https://ncbi.nlm.nih.gov/pubmed/29311663 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/s41467-017-02611-z |
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