Staphylococcus aureus produces pain through pore-forming toxins and neuronal TRPV1 that is silenced by QX-314

The hallmark of many bacterial infections is pain. The underlying mechanisms of pain during live pathogen invasion are not well understood. Here, we elucidate key molecular mechanisms of pain produced during live methicillin-resistant Staphylococcus aureus (MRSA) infection. We show that spontaneous...

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Pubblicato in:Nat Commun
Autori principali: Blake, Kimbria J., Baral, Pankaj, Voisin, Tiphaine, Lubkin, Ashira, Pinho-Ribeiro, Felipe Almeida, Adams, Kelsey L., Roberson, David P., Ma, Yuxin C., Otto, Michael, Woolf, Clifford J., Torres, Victor J., Chiu, Isaac M.
Natura: Artigo
Lingua:Inglês
Pubblicazione: Nature Publishing Group UK 2018
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Article
Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC5750211/
https://ncbi.nlm.nih.gov/pubmed/29295977
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/s41467-017-02448-6
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