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Inhibition of nitric oxide synthase unmasks vigorous vasoconstriction in established pulmonary arterial hypertension
It is widely accepted that impaired bioavailability of endothelial nitric oxide (NO) plays a critical role in the pathophysiology of pulmonary arterial hypertension (PAH). However, there are published data that show that relatively many PAH patients respond favorably to acetylcholine‐induced pulmona...
Tallennettuna:
| Julkaisussa: | Physiol Rep |
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| Päätekijät: | , , , , , , , , , |
| Aineistotyyppi: | Artigo |
| Kieli: | Inglês |
| Julkaistu: |
John Wiley and Sons Inc.
2017
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| Aiheet: | |
| Linkit: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5727286/ https://ncbi.nlm.nih.gov/pubmed/29208691 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.14814/phy2.13537 |
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