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Genetic or pharmacologic inhibition of EGFR ameliorates sepsis-induced AKI
Despite recent studies have demonstrated that the EGF receptor (EGFR) activation provided a renoprotective role during ischemic and folic acid-induced AKI, the role and regulation mechanism of EGFR in septic AKI remains unclear. Here, gefitinib, a highly selective EGFR inhibitor, abrogated LPS-induc...
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| Опубликовано в: : | Oncotarget |
|---|---|
| Главные авторы: | , , , , |
| Формат: | Artigo |
| Язык: | Inglês |
| Опубликовано: |
Impact Journals LLC
2017
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| Предметы: | |
| Online-ссылка: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5710948/ https://ncbi.nlm.nih.gov/pubmed/29207668 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.18632/oncotarget.21244 |
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