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Blood-brain barrier hyperpermeability precedes demyelination in the cuprizone model
In neuroinflammatory disorders such as multiple sclerosis, the physiological function of the blood-brain barrier (BBB) is perturbed, particularly in demyelinating lesions and supposedly secondary to acute demyelinating pathology. Using the toxic non-inflammatory cuprizone model of demyelination, we...
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| Опубликовано в: : | Acta Neuropathol Commun |
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| Главные авторы: | , , , , , , , , , |
| Формат: | Artigo |
| Язык: | Inglês |
| Опубликовано: |
BioMed Central
2017
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| Предметы: | |
| Online-ссылка: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5710130/ https://ncbi.nlm.nih.gov/pubmed/29195512 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/s40478-017-0497-6 |
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