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DRES-16. PARP INHIBITORS RESTORE TEMOZOLOMIDE SENSITIVITY IN MSH6-DEFICIENT TEMOZOLOMIDE-RESISTANT GLIOMA MODELS

INTRODUCTION: Mismatch repair (MMR) deficiency through MSH6 inactivation has been identified in approximately 30% of recurrent gliomas. This MMR deficiency represents a key molecular mechanism of acquired resistance to the alkylating chemotherapeutic agent temozolomide (TMZ). In addition, it is know...

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Pubblicato in:Neuro Oncol
Autori principali: Higuchi, Fumi, Wakimoto, Hiroaki, Cahill, Daniel
Natura: Artigo
Lingua:Inglês
Pubblicazione: Oxford University Press 2017
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Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC5692025/
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/neuonc/nox168.274
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