SALL4 suppresses PTEN expression to promote glioma cell proliferation via PI3K/AKT signaling pathway

Spalt-like transcription factor 4 (SALL4), a oncogene, is known to participate in multiple carcinomas, and is up-regulated in glioma. However, its actual role and underlying mechanisms in the development of glioma remain unclear. The present study explored the molecular functions of SALL4 in promoti...

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Bibliografiset tiedot
Julkaisussa:J Neurooncol
Päätekijät: Liu, Chuanjin, Wu, Haibin, Li, Yanyan, Shen, Liang, Yu, Renchun, Yin, Hongwei, Sun, Ting, Sun, Chunming, Zhou, Youxin, Du, Ziwei
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: Springer US 2017
Aiheet:
Laboratory Investigation
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC5663806/
https://ncbi.nlm.nih.gov/pubmed/28887597
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1007/s11060-017-2589-3
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