Lack of Contribution of p66shc and Its Mitochondrial Translocation to Ischemia-Reperfusion Injury and Cardioprotection by Ischemic Preconditioning

Whereas high amounts of reactive oxygen species (ROS) contribute to cardiac damage following ischemia and reperfusion (IR), low amounts function as trigger molecules in the cardioprotection by ischemic preconditioning (IPC). The mitochondrial translocation and contribution of the hydrogen peroxide-g...

詳細記述

保存先:
書誌詳細
出版年:Front Physiol
主要な著者: Boengler, Kerstin, Bencsik, Péter, Palóczi, János, Kiss, Krisztina, Pipicz, Márton, Pipis, Judit, Ferdinandy, Péter, Schlüter, Klaus-Dieter, Schulz, Rainer
フォーマット: Artigo
言語:Inglês
出版事項: Frontiers Media S.A. 2017
主題:
Physiology
オンライン･アクセス:https://ncbi.nlm.nih.gov/pmc/articles/PMC5633811/
https://ncbi.nlm.nih.gov/pubmed/29051737
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3389/fphys.2017.00733
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