تحميل...
Pharmacological hypothesis: Nitric oxide‐induced inhibition of ADAM‐17 activity as well as vesicle release can in turn prevent the production of soluble endothelin‐converting enzyme
Endothelin‐1 (ET‐1) and nitric oxide (NO) are two highly potent vasoactive molecules with opposing effects on the vasculature. Endothelin‐converting enzyme (ECE) and nitric oxide synthase (NOS) catalyse the production of ET‐1 and NO, respectively. It is well established that these molecules play a c...
محفوظ في:
| الحاوية / القاعدة: | Pharmacol Res Perspect |
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| المؤلفون الرئيسيون: | , , , |
| التنسيق: | Artigo |
| اللغة: | Inglês |
| منشور في: |
John Wiley and Sons Inc.
2017
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| الموضوعات: | |
| الوصول للمادة أونلاين: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5625149/ https://ncbi.nlm.nih.gov/pubmed/28971608 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1002/prp2.335 |
| الوسوم: |
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