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VEGF‐A(165)b protects against proteinuria in a mouse model with progressive depletion of all endogenous VEGF‐A splice isoforms from the kidney
KEY POINTS: Progressive depletion of all vascular endothelial growth factor A (VEGF‐A) splice isoforms from the kidney results in proteinuria and increased glomerular water permeability, which are both rescued by over‐expression of VEGF‐A(165)b only. VEGF‐A(165)b rescues the increase in glomerular b...
שמור ב:
| הוצא לאור ב: | J Physiol |
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| Main Authors: | , , , , , |
| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
John Wiley and Sons Inc.
2017
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| נושאים: | |
| גישה מקוונת: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5621502/ https://ncbi.nlm.nih.gov/pubmed/28574576 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1113/JP274481 |
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