Tunicamycin induced endoplasmic reticulum stress promotes apoptosis of prostate cancer cells by activating mTORC1

Studies suggest that tunicamycin may work as a therapeutic drug to cancer cells by inducing stress in the endoplasmic reticulum (ER) through unfolded protein response (UPR) and thereby promoting apoptosis. However, mechanisms of the prolonged activation of the UPR under sustained ER stress in the re...

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Kaydedildi:
Detaylı Bibliyografya
Yayımlandı:Oncotarget
Asıl Yazarlar: Guha, Prasun, Kaptan, Engin, Gade, Padmaja, Kalvakolanu, Dhananjaya V., Ahmed, Hafiz
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: Impact Journals LLC 2017
Konular:
Research Paper
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC5620248/
https://ncbi.nlm.nih.gov/pubmed/28978108
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.18632/oncotarget.19277
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