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Riboflavin supplementation does not attenuate hyperoxic lung injury in transgenic (spc–mt)hGR mice
The aims of this study were to test the hypothesis that mice expressing mitochondrially targeted human glutathione reductase (GR) driven by a surfactant protein C promoter ((spc–mt)hGR) are functionally riboflavin deficient and that this deficiency exacerbates hyperoxic lung injury. The authors furt...
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| Опубликовано в: : | Exp Lung Res |
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| Главные авторы: | , , , |
| Формат: | Artigo |
| Язык: | Inglês |
| Опубликовано: |
2010
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| Предметы: | |
| Online-ссылка: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5611852/ https://ncbi.nlm.nih.gov/pubmed/21128861 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3109/01902148.2010.516057 |
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