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Riboflavin supplementation does not attenuate hyperoxic lung injury in transgenic (spc–mt)hGR mice

The aims of this study were to test the hypothesis that mice expressing mitochondrially targeted human glutathione reductase (GR) driven by a surfactant protein C promoter ((spc–mt)hGR) are functionally riboflavin deficient and that this deficiency exacerbates hyperoxic lung injury. The authors furt...

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Bibliographic Details
Published in:Exp Lung Res
Main Authors: Heyob, Kathryn M., Rogers, Lynette K., Tipple, Trent E., Welty, Stephen E.
Format: Artigo
Language:Inglês
Published: 2010
Subjects:
Online Access:https://ncbi.nlm.nih.gov/pmc/articles/PMC5611852/
https://ncbi.nlm.nih.gov/pubmed/21128861
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3109/01902148.2010.516057
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