Guanylyl cyclase sensitivity to nitric oxide is protected by a thiol oxidation-driven interaction with thioredoxin-1

Nitric oxide (NO) modulates many physiological events through production of cGMP from its receptor, the NO-sensitive guanylyl cyclase (GC1). NO also appears to function in a cGMP-independent manner, via S-nitrosation (SNO), a redox-based modification of cysteine thiols. Previously, we have shown tha...

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Detalhes bibliográficos
Publicado no:J Biol Chem
Main Authors: Huang, Can, Alapa, Maryam, Shu, Ping, Nagarajan, Narayani, Wu, Changgong, Sadoshima, Junichi, Kholodovych, Vladyslav, Li, Hong, Beuve, Annie
Formato: Artigo
Idioma:Inglês
Publicado em: American Society for Biochemistry and Molecular Biology 2017
Assuntos:
Cell Biology
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC5582831/
https://ncbi.nlm.nih.gov/pubmed/28659344
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1074/jbc.M117.787390
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