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TRIM9-dependent ubiquitination of DCC constrains kinase signaling, exocytosis, and axon branching
Extracellular netrin-1 and its receptor deleted in colorectal cancer (DCC) promote axon branching in developing cortical neurons. Netrin-dependent morphogenesis is preceded by multimerization of DCC, activation of FAK and Src family kinases, and increases in exocytic vesicle fusion, yet how these oc...
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| Udgivet i: | Mol Biol Cell |
|---|---|
| Main Authors: | , , , , , , , |
| Format: | Artigo |
| Sprog: | Inglês |
| Udgivet: |
The American Society for Cell Biology
2017
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| Fag: | |
| Online adgang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5576901/ https://ncbi.nlm.nih.gov/pubmed/28701345 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1091/mbc.E16-08-0594 |
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