CaMKIIδ subtypes differentially regulate infarct formation following ex vivo myocardial ischemia/reperfusion through NF-κB and TNF-α

Deletion of Ca(2+/)calmodulin-dependent protein kinase II delta (CaMKIIδ) has been shown to protect against in vivo ischemia/reperfusion (I/R) injury. It remains unclear which CaMKIIδ isoforms and downstream mechanisms are responsible for the salutary effects of CaMKIIδ gene deletion. In this study...

Descrizione completa

Salvato in:
Dettagli Bibliografici
Pubblicato in:J Mol Cell Cardiol
Autori principali: Gray, Charles B.B., Suetomi, Takeshi, Xiang, Sunny, Mishra, Shikha, Blackwood, Erik A., Glembotski, Christopher C., Miyamoto, Shigeki, Westenbrink, B. Daan, Brown, Joan Heller
Natura: Artigo
Lingua:Inglês
Pubblicazione: 2017
Soggetti:
Article
Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC5564300/
https://ncbi.nlm.nih.gov/pubmed/28077321
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.yjmcc.2017.01.002
Tags: Aggiungi Tag
Nessun Tag, puoi essere il primo ad aggiungerne! !

Documenti analoghi