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CaMKIIδ Mediates Myocardial Ischemia/Reperfusion Injury Through NF-κB

RATIONALE: Ca(2+)/calmodulin-dependent protein kinase II (CaMKII) has been implicated as a maladaptive mediator of cardiac ischemic injury. We hypothesized that the inflammatory response associated with in vivo ischemia/reperfusion (I/R) is initiated through CaMKII signaling. OBJECTIVE: To assess th...

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Autori principali: Ling, Haiyun, Gray, Charles B.B., Zambon, Alexander C., Grimm, Michael, Gu, Yusu, Dalton, Nancy, Purcell, Nicole H., Peterson, Kirk, Brown, Joan Heller
Natura: Artigo
Lingua:Inglês
Pubblicazione: 2013
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Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC3673710/
https://ncbi.nlm.nih.gov/pubmed/23388157
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1161/CIRCRESAHA.112.276915
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