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FUNCTIONAL COMMUNICATION BETWEEN PKC-TARGETED CARDIAC TROPONIN I PHOSPHORYLATION SITES
Increased protein kinase C (PKC) activity is associated with heart failure, and can target multiple cardiac troponin I (cTnI) residues in myocytes, including S23/24, S43/45 and T144. In earlier studies, cTnI-S43D and/or -S45D augmented S23/24 and T144 phosphorylation, which suggested there is commun...
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| Izdano u: | Arch Biochem Biophys |
|---|---|
| Glavni autori: | , , , , |
| Format: | Artigo |
| Jezik: | Inglês |
| Izdano: |
2017
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| Teme: | |
| Online pristup: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5555030/ https://ncbi.nlm.nih.gov/pubmed/28587770 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.abb.2017.05.019 |
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