Cardiac myocyte p38α kinase regulates angiogenesis via myocyte-endothelial cell cross-talk during stress-induced remodeling in the heart

Stress-induced p38 mitogen-activated protein kinase (MAPK) activity is implicated in pathological remodeling in the heart. For example, constitutive p38 MAPK activation in cardiomyocytes induces pathological features, including myocyte hypertrophy, apoptosis, contractile dysfunction, and fetal gene...

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Detalhes bibliográficos
Publicado no:J Biol Chem
Main Authors: Rose, Beth A., Yokota, Tomohiro, Chintalgattu, Vishnu, Ren, Shuxun, Iruela-Arispe, Luisa, Khakoo, Aarif Y., Minamisawa, Susumu, Wang, Yibin
Formato: Artigo
Idioma:Inglês
Publicado em: American Society for Biochemistry and Molecular Biology 2017
Assuntos:
Cell Biology
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC5546022/
https://ncbi.nlm.nih.gov/pubmed/28637870
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1074/jbc.M117.784553
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