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Bcl-2 enhances Ca(2+) signaling to support the intrinsic regenerative capacity of CNS axons
At a certain point in development, axons in the mammalian CNS undergo a profound loss of intrinsic growth capacity, which leads to poor regeneration after injury. Overexpression of Bcl-2 prevents this loss, but the molecular basis of this effect remains unclear. Here, we report that Bcl-2 supports a...
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| Main Authors: | , , , , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
2005
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC554135/ https://ncbi.nlm.nih.gov/pubmed/15719013 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/sj.emboj.7600589 |
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