Helicobacter pylori modulates host cell responses by CagT4SS-dependent translocation of an intermediate metabolite of LPS inner core heptose biosynthesis

Highly virulent Helicobacter pylori cause proinflammatory signaling inducing the transcriptional activation and secretion of cytokines such as IL-8 in epithelial cells. Responsible in part for this signaling is the cag pathogenicity island (cagPAI) that codetermines the risk for pathological sequela...

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Publicado en:PLoS Pathog
Autores principales: Stein, Saskia C., Faber, Eugenia, Bats, Simon H., Murillo, Tatiana, Speidel, Yvonne, Coombs, Nina, Josenhans, Christine
Formato: Artigo
Lenguaje:Inglês
Publicado: Public Library of Science 2017
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Research Article
Acceso en línea:https://ncbi.nlm.nih.gov/pmc/articles/PMC5531669/
https://ncbi.nlm.nih.gov/pubmed/28715499
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1371/journal.ppat.1006514
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